1. Objective: Study the correlation between t cell anergy and apoptosis.
目的:探讨无能T细胞与凋亡的关系。

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2. Clonal anergy and suppressor cells are main mechanisms of the tolerance.
克隆不应答和抑制细胞是耐受的主要机制。

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3. To study the induced condition and characteristics of T cell anergy in vitro.
目的:在体外探讨T细胞无能诱导的条件及其生物学特性。

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4. Aim To explore the molecular mechanism of t cell anergy induced by superantigen SEA.
目的探讨超抗原诱导T细胞无能的分子机制。

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5. Regulatory DCs can induce regulatory T cells, inhibit T cell proliferation or induce T cell anergy.
目前发现,调节性DC可诱导调节性T细胞产生,抑制T细胞增殖或诱导T细胞增殖阻滞。

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6. The enhanced cell surface expression of CTLA-4 is likely responsible for the induction of t cell anergy.
结论CTLA - 4表面表达水平的升高很可能与T细胞无能状态的诱导有关。

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7. Introduces the concepts of available anergy and reduced exergy by analysing the energy utilization process.
通过对其能量利用过程的分析,引入可用和折合的概念。

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8. Howeer, some T cells are not actiated, in fact they are inactiated by a process called anergy or tolerance.
然而,有些T细胞处于未激发状态,事实上他们被称为无反应性,耐受性的过程灭活了。

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9. Howeer, some T cells are not actiated, in fact they are inactivated by a process called anergy or tolerance.
然而,有些T细胞处于未激发状态,事实上他们被称为无反应性,耐受性的过程灭活了。

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10. However, some t cells are not activated, in fact they are inactivated by a process called anergy or tolerance.
然而,有些T细胞处于未激发状态,事实上他们被称为无反应性,耐受性的过程灭活了。

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11. CD47 induces costimulatory signals on activation of T cell, T cell apoptosis and T cell anergy and enhances the efficiency of TCR signaling;
CD4 7是T细胞活化的共刺激因子,活化T细胞凋亡过程,诱导T细胞无反应性,加强TCR信号传导的效率;

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12. Several other mechanisms may by theorized for the induction of tolerance in this renal transplant model, including clonal anergy and suppressive cells.
结论嵌合状态与肾移植耐受存在明显相关性,克隆不应答、抑制细胞参与了耐受的形成。

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13. The IL2 reverse test and detection of microchimerism showed that the tolerance mechanism involved anergy, and was related to formation of microchimerism.
IL 2逆转实验、微嵌合体检测表明,该耐受与克隆失活、微嵌合体的存在有关。

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14. Given that severe trauma effects as many as 20 percent of the 20,000 or so human genes, finding the genes and proteins involved in t cell anergy and apoptosis amid this storm has been a challenge.
如果大面积损伤影响多达人类基因(大概20000个)中20%的基因的话,找到其中涉及T细胞无能和凋亡的基因和蛋白是一个挑战。

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15. Given that severe trauma effects as many as 20 percent of the 20,000 or so human genes, finding the genes and proteins involved in t cell anergy and apoptosis amid this storm has been a challenge.
如果大面积损伤影响多达人类基因(大概20000个)中20%的基因的话,找到其中涉及T细胞无能和凋亡的基因和蛋白是一个挑战。

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