1. Survival duration of mice after cerebral anoxia.
脑缺氧后小鼠存活时间。
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2. Results The main cause of acute encephalocele were delayed intracranial hematomas, acute diffuse brain swelling, cerebral contusion and laceration in Sylvain tissue and ischemic anoxia.
3. BACKGROUND: After cerebral tissue ischemia and anoxia in young rats, the cerebral edema gets serious, and the levels of nitric oxide (no) and malondialdehyde (MDA) decrease.
背景：幼鼠脑缺氧缺血后，脑组织水肿加重，脑组织中一氧化氮及丙二醛水平增高。
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4. Objective To observe the dynamic change of nitric oxide (no) contents in the cerebral tissues of mices of anoxia asphyxia.
目的研究小鼠缺氧窒息后脑组织一氧化氮(NO)含量的动态变化。
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5. Objective:To study the effects of grape seed proanthocyanidins(GSP) on injuries of cerebral ischemia and reperfusion and anoxia in mice.
目的：研究葡萄籽原花青素（GSP）对小鼠脑缺血、再灌注损伤及常压缺氧的影响。
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6. Methods (1) The experiments of quick decapitation and anoxia under normal pressure model in mice was used to study the effects of LW on acute cerebral ischemia and anoxia.
方法：（1）采用小鼠断头实验和常压耐缺氧实验，观察LW对脑组织急性缺血缺氧的保护作用；
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7. MethodsThe general level experiment was applied acute cerebral ischemia or anoxia experimental method.
整体水平实验部分采用急性脑缺血缺氧的实验方法。
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8. Results: Extract liquid Qishengwan could obviously improve the protective effect on the mice anoxia, revive the SOD activity of the cerebral tiss.
目的：观察七圣丸水提物抗缺氧与抗脂质过氧化作用。
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9. These results indicated that squalene could protect mice from anoxia caused by acute cerebral ischemia and sodium nitrite poisoning.
表明角鲨烯对急性脑缺血性和亚硝酸钠中毒缺氧具有保护作用。
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10. These results indicated that squalene could protect mice from anoxia caused by acute cerebral ischemia and sodium nitrite poisoning.
